Oxidative stress arises from an imbalance between reactive oxygen species (ROS) production and antioxidant defenses, damaging DNA, proteins, and lipids while also modulating immune responses. It plays a central role in neurodegeneration, cancer, chronic fatigue syndrome, and post-viral conditions like Long COVID, with mitochondrial dysfunction and chronic inflammation amplifying redox imbalance. Therapeutic strategies—including antioxidants, Nrf2 activators, lifestyle interventions, and biomarker-guided approaches—aim to restore redox homeostasis, improve cellular function, and mitigate disease progression.
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- Tobias Giesen (Autor:in), 2024, Oxidative Stress and Redox Balance Biochemical Mechanisms, Clinical Relevance, and Therapeutic Perspectives, München, GRIN Verlag, https://www.grin.com/document/1619706
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